faculty

Andy Wessels Grant Support

Active

1R01HL122906-01A1 Role: Principal Investigator 09/01/15-08/31/18

NIH-HLBI
Mechanisms of DMP Development and Atrioventricular Septation
Goal of project: To investigate the synergistic relationship between Shh, BMP and Wnt/beta-catenin pathways and primary cilia in the regulation of DMP development and in the formation of the AV septal complex.


13GRNT 16220004 Role: Principal Investigator 07/01/13-06/30/15

American Heart Association
The Epicardium and Atrioventricular Valve Development
The major goal of the project is to further determine the role of the epicardium and epicardially-derived cells in the formation and maturation of the leaflets of the AV valves. 


1R56 HL122906Role: Principal Investigator09/01/14-08/31/15

NIH-HLBI
Mechanisms of DMP Development and Atrioventricular Septation
The major goal of this project is to test the hypothesis that primary cilia are functioning as a signaling center in the pSHF/DMP and are critically important in the development of the DMP and in atrioventricular septation. 


1P30 GM103342-03 Role:  Program Coordinator 08/01/12-04/30/17

NIH-NIGMS 
SC COBRE for Developmentally Based Cardiovascular Diseases (Phase III)
This COBRE Phase III renewal is aimed at sustaining the research infrastructure built during the first 10 years of NCRR funding. 


5R01 HL033756 Role: Co-Investigator04/01/87-05/31/16

NIH NHLBI
Mucopolysaccharide Metabolism in Cardiac Anomalies
The major goals of this project are to determine if the heart defect (htd) gene is a prototype candidate for an embryonic segmentation gene; to determine if the fate of the luminal “face” of cushion mesenchyme is to expand into the definitive precursor valve leaflets; and to determine if a primary fate of cushions is to interact with the myocardium to complete septal development. 


1R01HL121382 Role: Co-Investigator 09/01/14-06/30/18

NIH-HLBI 
Proteoglycan Regulation during Cardiac Valve Development and Homeostasis
The major goal of this project is to investigate the interconnection of extracellular matrix remodeling, with cell behavior and signaling an area of valve development and disease.


Pending

 

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